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Addiction Psychiatry Pass Rates

Addiction Psychiatry Board Exam Prep

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  • Earn up to 137 AMA PRA Category 1 Credits™, including 24 ABPN-Approved Self-Assessment Category 1 Credits
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What are the 2024 Addiction Psychiatry Board Pass Rates?

The ABPN 2024 Addiction Psychiatry Pass Rate for the Certification exam is 95%.
The ABPN 2024 Addiction Psychiatry Pass Rate for the Continuing Certification exam is 100%.

Compare to exam takers who prepared with Beat The Boards!

In 2024, Beat The Boards! Addiction Psychiatry Board Review clients achieved a 95% pass rate on the Certification exam and a 100% pass rate on the CC exam!

Why take chances? Beat The Boards! Addiction Medicine Board Review GUARANTEES you’ll pass!

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“One of the best courses in the country. Key to success.” - Shah M. Nadeem, MD, Gaithersburg, MD

“I passed the examination. I would not have done it without your course.” - Umnmaheswara Kalapatapu, MD, Logansport, IN

“Useful not only for Board Preparation but in daily clinical practice for patient care and prescribing. The course has been very stimulating to me for passing my boards...” - Mahmooda, MD, Hafiz, MD

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What do I do if I fail the Addiction Psychiatry Examination?

The first thing you should do is just take it easy, sleep on it. Give yourself a few days, a week or two to come to terms with what has happened. Your next exam is six months to a year away. Dr. Jack has more advice in this video, What Do You Do If You Fail Your Medical Board Exam?

ABPN Addiction Psychiatry Examination Scoring

There is no predefined passing rate for Addiction Psychiatry examinees. Each examinee should have some degree of familiarity with the subject matter of each question. Even though the examinee may be in doubt about the correct answer to a particular question, they should answer every question. This will increase the likelihood that the examinee’s examination score will reflect the breadth of their knowledge of the field. There is no penalty for guessing.

ABPN Board Examination Format

The computer-delivered Addiction Psychiatry Continuing Certification Examination primarily consists of 170 test questions to be completed within 170 minutes (2 hours and 50 minutes). The Addiction Psychiatry Continuing Certification Examination consists of single-best-answer multiple-choice questions. Example of a single-best-answer multiple-choice question format:

A 44-year-old woman presents with an alcohol use disorder. She tells you she previously failed treatment with disulfiram prescribed by her internist. You prescribe naltrexone, and after 8 weeks of treatment, she happily reports a significant decrease in alcohol intake. You recall studying that people with this certain gene variant respond better to naltrexone treatment. Which allele would cause a patient to respond better to this treatment?

◯ A. Dopamine beta–hydroxylase ◯ B. Human dopamine D2 receptor E8 A/A ◯ C. Ankyrin repeat and kinase domain containing 1 rs1800497 ◯ D. Dopamine active transporter 1 10/10 repeat homozygotes ◯ E. Opioid receptor mu 1 A118G

Hover here for correct answer. The correct answer is: E. Opioid receptor mu 1 A118G Presently, the US Food and Drug Administration has approved the use of disulfiram, naltrexone, and acamprosate for the treatment of AUD. One of the most constant results of naltrexone has been the decline in heavy drinking relapse, heavy drinking days, overall drinking, and alcohol craving. It has been noted that therapeutic responses to naltrexone in AUD are regulated by variations at the mu-opioid receptor gene locus (OPRM1). There are certain alcohol-dependent individuals who respond better to naltrexone treatment and they would be carriers of the OPRM1 118G allele. People low on dopamine beta-hydroxylase exhibit lack of disulfiram effects on their cocaine use while those with the Human dopamine D2 receptor E8 A/A genotype were noted to need higher doses of tiapride for alcohol use disorder, as well as increased depression and anxiety upon admission and 2 weeks after admission. Carriers of the ANKK1 rs1800497 T allele showed fewer cocaine-positive urines during the disulfiram pharmacotherapy while dopamine active transporter 1 10/10 repeat homozygotes do not reduce drinking in response to naltrexone. Source Patriquin MA, Bauer IE, Soares JC, Graham DP, Nielsen DA. Addiction pharmacogenetics. Psychiatric Genetics. 2015;25(5):181-193. doi:10.1097/ypg.0000000000000095. Bilbao A, Robinson JE, Heilig M, et al. A Pharmacogenetic Determinant of Mu–Opioid Receptor Antagonist Effects on Alcohol Reward and Consumption: Evidence from Humanized Mice. Biological Psychiatry. 2015;77(10):850-858. doi:10.1016/j.biopsych.2014.08.021. Medications Development Program. November 2017. https://www.niaaa.nih.gov/research/major–initiatives/medications–development–program. Sub-Topic Genetics
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